Resume

• 2018

  Columbia University School of Professional Studies

  Farmingdale State College

  Columbia University School of Professional Studies

  Stony Brook University

  Stony Brook

  NY

  My dissertation research focused on the impacts that lifestyle factors have on cognitive decline and associated pathologies in transgenic murine models of Alzheimer’s disease. I used a wide range of behavioral and biochemical techniques in order to investigate potential cognitive

  pathological and neuronal changes that follow various forms of environmental interventions.

  PhD Candidate - Integrative Neuroscience

  Farmingdale State College

• 2012

  Italian

  English

  Doctor of Philosophy (Ph.D.)

  Integrative Neuroscience

  Stony Brook University

• 2010

  Master of Science - MS

  Psychology

  Stony Brook University

• 2006

  Bachelor of Arts (BA)

  Psychology

  Italian Language and Literature

  Stony Brook University

• New York State Foundation for Science

  Technology and Innovation

  Volunteer

  Alzheimer's Association

  New York City Chapter

  New Orleans Area Habitat for Humanity

  Public Speaking

  Behavioral Neuroscience

  Transgenic Mice

  Teaching

  ELISA

  College Teaching

  Science

  Immunohistochemistry

  Scientific Writing

  Psychology

  Research

  Alzheimer's Disease

  Experimental Design

  Data Analysis

  Statistics

  Neuroscience

  PowerPoint

  Histology

  Microsoft Office

  SPSS

  Galanin

  through GalR1 but not GalR2 receptors

  decreases motivation at times of high appetitive behavior.

  John K. Robinson

  Ulo Langel

  Galanin is a 29/30-amino acid long neuropeptide that has been implicated in many physiological and behavioral functions. Previous research has shown that i.c.v. administration of galanin strongly stimulates food intake in sated rats when food is freely available

  but fails to stimulate this consumption when an operant response requirement is present. Using fixed ratio (FR) schedules

  we sought to further clarify galanin's role in motivated behavior by administering galanin i.c.v. to rats working on fixed ratio schedules requiring either a low work condition (FR1) or higher work conditions (FR > 1) to obtain a 0.2% saccharin reward. Rats in the FR > 1 group were assigned to either an FR3

  FR5 or FR7 schedule of reinforcement. The rate of reinforcement decreased for only the FR > 1 group as compared to saline controls. Furthermore

  injections of GalR1 receptor agonist M617 led to a similar

  marginally significant decrease in the number of reinforcers received in the FR > 1 condition

  but a decrease was not seen after injections of GalR2 receptor agonist M1153. Taken together

  these results show that galanin may be playing a role in decreasing motivation at times of high appetitive behavior

  and that this effect is likely mediated by the GalR1 receptor.

  Galanin

  through GalR1 but not GalR2 receptors

  decreases motivation at times of high appetitive behavior.

  Cerebral Microvascular Rather than Parenchymal Amyloid-β Protein Pathology Promotes Early Cognitive Impairment in Transgenic Mice

  Anderson

  Stony Brook University